Loss of consciousness when coughing in men - About blood vessels
If a patient is concerned about episodes of loss of consciousness due to a cough attack or any other pathology, he should be examined by a neurologist. Additionally, a consultation and examination with a pulmonologist or cardiologist may be prescribed.
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General information
The term “bettolepsy” was first proposed by the Soviet neurologist M.I. Kholodenko in 1941 for the interpretation of paroxysms that occur at the height of coughing attacks. The pathology is observed quite rarely, accounting for no more than 2% of cases of all types of paroxysmal conditions.
Bettolepsy can occur under the names “cough-brain syndrome”, “cough syncope”, “laryngeal vertigo”, “respiratory seizure”, “cough syncope”. It is more often observed in persons with symptoms of pulmonary heart failure. Mostly men aged 45 years and older are affected.
Pathogenesis
The pathogenesis of bettolepsy is not fully understood. Typically, paroxysmal conditions that occur at the height of the cough reflex have nothing to do with epilepsy. The hemodynamic theory explains most fully the changes that occur during coughing. There are three phases of cough: inspiratory, compressive and expiratory.
In the compressive and expiratory phases, intrathoracic and intra-abdominal pressure increases sharply, resulting in a decrease in blood flow to the heart.
This leads to a decrease in cardiac output and changes in cerebrospinal fluid pressure in the brain and spinal cord.
As a result of a sharp increase in intrathoracic pressure, it increases in the peripheral arteries, veins and chambers of the heart, which leads to venous stagnation and causes bettolepsy.
There are other development mechanisms: stimulation of vagus nerve receptors, transmission of pathological impulses from reflexogenic areas of the respiratory tract and jugular veins. This kind of influence leads to changes in the functioning of the reticular formation, which is fraught with vasodepressor reactions and severe bradycardia with impaired consciousness.
Bettolepsy manifests itself against the background of hypoxia of brain tissue, both acute and chronic. Its immediate cause is a sharp worsening of an existing oxygen deficiency, provoked by a cough attack.
This condition manifests itself in the following pathologies:
- Chronic lung diseases, which include emphysema, asthma, cor pulmonale, tuberculosis. With these diseases, congestion in the pulmonary circulation is observed, and pulmonary heart failure then progresses. In case of decompensation, encephalopathy with a tendency to bettolepsy is provoked.
- Obstruction of the airways as a result of aspiration of foreign bodies, whooping cough, acute laryngitis. The condition is accompanied by brain hypoxia and prolonged coughing attacks, causing respiratory attacks.
- Cerebrovascular disorders: vascular malformations, compression of extracranial and intracranial veins, consequences of traumatic brain injuries, pathologies of intra- and extracranial arteries. Because of this, venous hyperemia of the brain develops, which can be accompanied by cough syncope.
- Lesions of the peripheral parts of the nervous system. Neuralgia of the superior laryngeal nerve due to pathological impulses leads to activation of the center of the vagus nerve, which causes bradycardia. Cardiac output decreases, cerebral ischemia and fainting occur.
The list of risk factors that are important in the progression of attacks of bettolepsy should include excess body weight, smoking, alcohol abuse, and drug use.
The pathogenesis of this condition is not thoroughly understood. Most often, paroxysms that occur at the height of a coughing attack have no common features with epilepsy. The mechanism of changes is most fully described by the hemodynamic theory.
There are three phases of cough: inspiratory, compressive, expiratory. In the second and third phases, there is a sharp increase in intrathoracic and intra-abdominal pressure, which reduces blood flow to the heart.
Because of this, cardiac output decreases and cerebrospinal fluid pressure in the spinal cord and brain changes. Due to a sharp increase in intrathoracic pressure, it increases in the peripheral vessels and chambers of the heart, which provokes venous stagnation, and then betolepsy develops.
Another mechanism for the development of the condition is also possible: as a result of stimulation of the vagus nerve receptors, pathological impulses are transmitted from the reflexogenic areas of the airways and jugular veins. Because of this, the functionality of the reticular formation changes, and this causes vasodepressor reactions, severe bradycardia and disturbances of consciousness.
The pathogenesis of bettolepsy is not fully understood. Typically, paroxysmal conditions that occur at the height of the cough reflex have nothing to do with epilepsy. The hemodynamic theory explains most fully the changes that occur during coughing. There are three phases of cough: inspiratory, compressive and expiratory.
In the compressive and expiratory phases, intrathoracic and intra-abdominal pressure increases sharply, resulting in a decrease in blood flow to the heart.
This leads to a decrease in cardiac output and changes in cerebrospinal fluid pressure in the brain and spinal cord.
As a result of a sharp increase in intrathoracic pressure, it increases in the peripheral arteries, veins and chambers of the heart, which leads to venous stagnation and causes bettolepsy.
There are other development mechanisms: stimulation of vagus nerve receptors, transmission of pathological impulses from reflexogenic areas of the respiratory tract and jugular veins. This kind of influence leads to changes in the functioning of the reticular formation, which is fraught with vasodepressor reactions and severe bradycardia with impaired consciousness.
Classification
Bettolepsy syndrome has not been fully studied. Despite the high prevalence of diseases and conditions accompanied by cough, this symptom complex is rare. Its course can be grouped according to clinical manifestations:
- Short-term twilight disorder of consciousness. Usually lasts a few seconds and does not require emergency treatment. In this case, the underlying disease that caused the condition should be treated.
- Brief fainting at the height of coughing. Most often it lasts from 2 to 10 seconds. Treatment of the underlying pathology is necessary.
- Prolonged loss of consciousness. Complicated by convulsions, involuntary urination, defecation. Often combined with organic brain damage with lasting consequences. Aggravating factors include alcohol, nicotine intoxication, and drug poisoning.
Bettolepsy syndrome has not been fully studied. Despite the high prevalence of diseases and conditions accompanied by cough, this symptom complex is rare. Its course can be grouped according to clinical manifestations:
- Brief twilight disorder of consciousness. Usually lasts a few seconds and does not require emergency treatment. In this case, the underlying disease that caused the condition should be treated.
- Brief fainting at the height of coughing. Most often it lasts from 2 to 10 seconds. Treatment of the underlying pathology is necessary.
- Prolonged loss of consciousness. Complicated by convulsions, involuntary urination, defecation. Often combined with organic brain damage with lasting consequences. Aggravating factors include alcohol, nicotine intoxication, and drug poisoning.
1. Short-term twilight disorder of consciousness. Usually lasts a few seconds and does not require emergency treatment. In this case, the underlying disease that caused the condition should be treated.
2. Short fainting at the height of coughing. Most often it lasts from 2 to 10 seconds. Treatment of the underlying pathology is necessary.
3. Prolonged loss of consciousness. Complicated by convulsions, involuntary urination, defecation. Often combined with organic brain damage with lasting consequences. Aggravating factors include alcohol, nicotine intoxication, and drug poisoning.
Complications
All people, without exception, cough. Some people get scared at the first cough and start taking antibiotics, while others, on the contrary, do not pay attention to the symptom for a long time.
But few of them know that a strong cough can lead to unpleasant consequences: from loss of consciousness to pathological changes in the brain. This type of attack is called bettolepsy.
Its manifestations should never be ignored; it is important to consult a doctor as soon as possible.
Complications rarely occur with bettolepsy. They are usually associated with the underlying disease that caused the syndrome. One of the serious consequences is increasing pulmonary heart failure.
Circulatory disorders in the brain can lead to permanent damage to cerebral tissue - hypoxic encephalopathy.
During cough syncope, there is a risk of injury if you fall from your own height.
But few of them know that a strong cough can lead to unpleasant consequences: from loss of consciousness to pathological changes in the brain. This type of attack is called bettolepsy.
Its manifestations should never be ignored; it is important to consult a doctor as soon as possible.
Complications rarely occur with bettolepsy. They are usually associated with the underlying disease that caused the syndrome. One of the serious consequences is increasing pulmonary heart failure.
Circulatory disorders in the brain can lead to permanent damage to cerebral tissue - hypoxic encephalopathy.
During cough syncope, there is a risk of injury if you fall from your own height.
Prognosis and prevention
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Source: https://nevrolog-info.ru/poterya-soznaniya-kashle/
Bettolepsy
Bettolepsy
is a transient disturbance of consciousness that occurs at the peak of a coughing attack. The syndrome is manifested by cough syncope: short-term twilight consciousness, fainting or deep loss of consciousness, sometimes accompanied by convulsions, involuntary urination and defecation.
Methods for diagnosing bettolepsy include questioning, examination of the patient, functional tests, instrumental studies (electrocardiography, electroencephalography, bronchoscopy).
Treatment involves symptomatic therapy that alleviates the patient’s condition and is aimed at eliminating the manifestations of the underlying disease.
The term “bettolepsy” was first proposed by the Soviet neurologist M.I. Kholodenko in 1941 for the interpretation of paroxysms that occur at the height of coughing attacks. The pathology is observed quite rarely, accounting for no more than 2% of cases of all types of paroxysmal conditions.
Bettolepsy can occur under the names “cough-brain syndrome”, “cough syncope”, “laryngeal vertigo”, “respiratory seizure”, “cough syncope”. It is more often observed in persons with symptoms of pulmonary heart failure.
Mostly men aged 45 years and older are affected.
Bettolepsy
The condition occurs against the background of acute or chronic hypoxia of brain tissue. Its immediate cause is a sharp worsening of an already existing lack of oxygen caused by coughing paroxysm. Pathology can manifest itself in the following diseases:
- Chronic pulmonary pathologies
(cor pulmonale, asthma, tuberculosis, emphysema). With these diseases, stagnation occurs in the pulmonary circulation, and pulmonary heart failure subsequently develops. In a decompensated course, the development of encephalopathy with a tendency to convulsive fainting is possible. - Airway obstruction
(aspiration of foreign bodies, whooping cough, acute laryngitis). Accompanied by acute cerebral hypoxia and prolonged bouts of severe coughing, which cause episodes of cough syncope. - Cerebrovascular disorders
. Changes in cerebral vessels (vascular malformations, compression of intracranial and extracranial veins, consequences of head injury) cause venous hyperemia of the brain, which can be accompanied by fainting attacks. Disturbances in the blood supply to the brain due to pathology of extra- and intracranial arteries (cerebral atherosclerosis, vertebral artery syndrome) threaten the development of a number of vestibular disorders, including loss of consciousness. - Lesions of peripheral nerves
. With neuralgia of the superior laryngeal nerve, pathological impulses lead to activation of the vagus nerve center and bradycardia. The volume of cardiac output sharply decreases, cerebral ischemia and fainting occur.
Risk factors for the development of attacks of impaired consciousness are smoking, drug addiction, and excess weight. When intoxicated with alcohol and drugs, changes occur in the brain, its membranes and cerebrospinal fluid, leading to disruption of the respiratory and cardiovascular systems.
The pathogenesis of bettolepsy is not fully understood. Typically, paroxysmal conditions that occur at the height of the cough reflex have nothing to do with epilepsy. The hemodynamic theory explains most fully the changes that occur during coughing. There are three phases of cough: inspiratory, compressive and expiratory.
In the compressive and expiratory phases, intrathoracic and intra-abdominal pressure increases sharply, resulting in a decrease in blood flow to the heart. This leads to a decrease in cardiac output and changes in cerebrospinal fluid pressure in the brain and spinal cord.
As a result of a sharp increase in intrathoracic pressure, it increases in the peripheral arteries, veins and chambers of the heart, which leads to venous stagnation and causes bettolepsy.
There are other development mechanisms: stimulation of vagus nerve receptors, transmission of pathological impulses from reflexogenic areas of the respiratory tract and jugular veins. This kind of influence leads to changes in the functioning of the reticular formation, which is fraught with vasodepressor reactions and severe bradycardia with impaired consciousness.
Bettolepsy syndrome has not been fully studied. Despite the high prevalence of diseases and conditions accompanied by cough, this symptom complex is rare. Its course can be grouped according to clinical manifestations:
- Brief twilight disorder of consciousness
. Usually lasts a few seconds and does not require emergency treatment. In this case, the underlying disease that caused the condition should be treated. - Brief fainting at the height of coughing
. Most often it lasts from 2 to 10 seconds. Treatment of the underlying pathology is necessary. - Prolonged loss of consciousness
. Complicated by convulsions, involuntary urination, defecation. Often combined with organic brain damage with lasting consequences. Aggravating factors include alcohol, nicotine intoxication, and drug poisoning.
Clinical manifestations may differ not only in different patients, but also each attack in an individual patient can take on different course options. Paroxysmal conditions - cough syncope - occur at the peak of the cough reflex.
Similar symptom complexes are also observed when laughing, sneezing, straining, lifting heavy objects, etc.
They may be preceded by prodromal phenomena (presyncopal states) in the form of dizziness, tinnitus, blurred vision, facial flushing, which is subsequently replaced by cyanosis, and swelling of the neck veins when coughing. In some cases, some warning signs may be missing.
Bettolepsy is accompanied by attacks of severe convulsive cough, at the height of which signs of impaired consciousness or fainting appear. Typically, the occurrence of an attack is not related to the position of the body.
A cough can be triggered by a strong smell or cold air. The duration of twilight consciousness or deep fainting ranges from a few seconds to 2-5 minutes.
At the peak of the cough, loss of consciousness is usually accompanied by a fall; most often, patients come to their senses without outside help.
Sometimes bettolepsy can be accompanied by convulsions that are local in nature: for example, twitching of the upper or lower extremities. The skin acquires a grayish-bluish tint, and profuse sweating appears.
Biting the tongue during an attack is usually not observed. In rare cases, bettolepsy leads to urinary and fecal incontinence.
With organic brain lesions, cough syncope can be replaced by petit epileptic seizures, which are not dependent on coughing.
In the post-syncope period, neck pain and headache may be felt. The patient complains of general weakness and dizziness, which disappear over time. The state of stupor and memory loss observed during epileptic attacks are not characteristic of bettolepsy. In the absence of aggravating factors, the consequences do not cause mental disorders.
Complications rarely occur with bettolepsy. They are usually associated with the underlying disease that caused the syndrome. One of the serious consequences is increasing pulmonary heart failure.
Circulatory disorders in the brain can lead to permanent damage to cerebral tissue - hypoxic encephalopathy.
During cough syncope, there is a risk of injury if you fall from your own height.
To make a correct diagnosis, a comprehensive clinical and instrumental examination is required to identify the cause of cough syncope, as well as differentiate it from other diseases. The diagnostic algorithm includes:
- Consultations with specialists
(therapist, neurologist, pulmonologist, cardiologist). At the appointment, the history of the disease, the nature of the attacks, and their connection with cough are studied. Great importance is attached to physical methods. During the examination, attention is paid to the general condition of the patient, constitutional features (tendency to obesity). - Vagal tests
(Valsalva maneuver, test with pressure on the carotid sinus). They are carried out with the aim of modeling the pathogenetic mechanisms of syncope. - EPI of the cardiovascular system.
An ECG allows you to identify pathological processes in the heart, indicating the presence of pulmonary heart failure. In some cases, stress tests and daily ECG monitoring are used. - EEG
. It makes it possible to record pathological impulses emanating from certain areas of the brain, which is extremely important for excluding organic cerebral lesions. Functional tests are used to identify foci of seizure activity. - Methods for assessing the bronchopulmonary system
(radiation diagnostics, endoscopy of the respiratory tract). X-ray of the lungs is used to identify chronic diseases of the respiratory system and pulmonary heart. With the help of tracheobronchoscopy, foreign bodies of the trachea and bronchi are detected and removed.
When carrying out differential diagnosis, loss of consciousness due to orthostatic hypotension, cerebral vascular occlusion, or epilepsy should be excluded. Episodes of loss of consciousness in these conditions are in no way related to the cough reflex.
During an attack, at the stage of pre-medical care for the patient, it is necessary to ensure the flow of arterial blood enriched with oxygen to the brain. For this purpose, it is necessary to lay the patient on his back, lower his head and raise his lower limbs, ensure free breathing and access to fresh air.
Medical care consists of measures aimed at reducing congestion in the brain, eliminating disturbances in the functioning of the cardiovascular system by administering cardiotonics, vasoconstrictors, as well as drugs that improve bronchial patency. For bradycardia, atropine is administered. Subsequently, the patient may be hospitalized in the department of neurology or pulmonology for treatment of the underlying disease.
To prevent paroxysmal conditions, it is necessary to monitor your health and, if symptoms of bettolepsy occur, promptly seek medical help. Diet is of great importance, since excess body weight is one of the risk factors.
It is necessary to avoid conditions that contribute to the development of fainting: prolonged cough, overwork, prolonged standing, severe tension, sudden movements of the head.
Good rest, gymnastics and sports, and hardening have a beneficial effect on the body.
Source: https://www.KrasotaiMedicina.ru/diseases/zabolevanija_neurology/betolepsy
What is the danger of bettolepsy: symptoms, treatment, complications
All people, without exception, cough. Some people get scared at the first cough and start taking antibiotics, while others, on the contrary, do not pay attention to the symptom for a long time.
But few of them know that a strong cough can lead to unpleasant consequences: from loss of consciousness to pathological changes in the brain. This type of attack is called bettolepsy.
Its manifestations should never be ignored; it is important to consult a doctor as soon as possible.
Why does an attack occur?
The origin of bettolepsy is related to:
- with incorrect impulses that come from the nerves to the cough center;
- with pathological perception of information in the reflex zones of the respiratory tract.
This leads to disturbances in the autonomic nervous system and excitation of the tenth pair of cranial nerves (vagus), causing severe bradycardia.
During intense coughing, hyperventilation of the lungs occurs and intrathoracic pressure increases. Because of this, cerebral circulation is disrupted and various disorders occur: short-term loss of consciousness, convulsions, amnesia, severe headache.
Read about the causes of sudden loss of consciousness and first aid for the victim.
How cerebral vasoconstriction manifests itself: symptoms and complications of severe vascular spasm.
There is only one provoking factor – cough. But there can be many risks:
- diseases of the lungs and bronchi - tuberculosis, asthma, emphysema, chronic bronchitis, whooping cough;
- foreign body entering the respiratory tract;
- inflammation of the laryngeal nerve;
- pathologies of cerebral vessels - atherosclerosis, compression of the arteries due to osteochondrosis;
- alcohol and tobacco abuse;
- diseases of the cardiovascular system - cor pulmonale, venous stagnation of blood;
- unhealthy diet, unhealthy lifestyle.
It is extremely important to know the cause of the development of bettolepsy, since the severity of the attack and treatment largely depend on this.
How does bettolepsy manifest?
The severity of bettolepsy symptoms may vary not only between different patients, but also within the same patient at different times. The disease attacks mainly older men; it can develop in children due to a cough, the cause of which is whooping cough. There are several variants of a cough-brain attack:
- The attack occurs at the peak of a strong cough. The patient loses consciousness and falls.
- Bettolepsy develops into epileptic seizures. They can already occur without a cough.
- Attacks that are accompanied by profound disturbances in the autonomic nervous system. Most often they occur in people with brain pathologies.
- Loss of consciousness is accompanied by convulsions, involuntary urination and defecation.
- History of seizures in patients with epilepsy.
Sometimes you can recognize the onset of bettolepsy and prevent the patient from falling:
- the face acquires a red and then bluish tint;
- lips become purple;
- the neck veins swell and pulsate greatly;
- the patient complains of dizziness.
Usually, loss of consciousness occurs before the end of the first minute of the attack, the patient stops coughing, falls and suddenly turns pale. If the patient does not have severe concomitant diseases, then consciousness resumes after a few minutes or even seconds. Most often, such patients do not require medical care.
After bettolepsy, memory disorders (amnesia), unpleasant, painful sensations in the neck, and headaches occur. Since an attack does not develop without an intense cough, it is important to know the factors that cause coughing:
- strong, prolonged laughter;
- inhalation of cold or hot air;
- sneeze;
- cigarette smoke or other irritating odors;
- smoking;
- lifting something heavy;
- the act of defecation during constipation.
How to diagnose the disease
If similar symptoms are detected, the patient should consult a family doctor or neurologist. To make a diagnosis, you need to carefully collect anamnesis, study the medical history and draw up the correct examination plan. It is important to differentiate bettolepsy from similar diseases, for example, epilepsy.
To determine brain-cough syndrome, the following examination methods are used:
- Holter monitoring – recording of a cardiogram during the day. Allows you to evaluate the work of the heart in the body’s usual conditions, as well as the reaction to various situations. Helps determine the cause of loss of consciousness.
- Tracheobronchoscopy is an endoscopic examination of the respiratory tract. The condition of the mucous membrane, the presence of foreign bodies, and the diameter of the bronchial lumens are determined.
- Valsalva maneuver - helps to assess the state of the autonomic nervous system. The patient should exhale all the air, then inhale deeply and exhale again, holding the breath for at least 15 seconds.
- ECG.
- ECHO-KG.
To make a diagnosis, hospitalization in an inpatient department is not always necessary. Most often, the patient comes for examinations. The exception is severe cough fainting with severe convulsions. In this case, the patient may be referred to a specialized epileptology center to clarify the disease.
How to treat bettolepsy
Treatment for bettolepsy, like most other diseases, is aimed at getting rid of the cause of the attacks. Therefore, it is prescribed individually after a thorough examination. After an attack, symptomatic therapy is carried out using:
- ammonia;
- cardiotonic drugs;
- saturating the body with oxygen;
- vasoconstrictor medications;
- for severe bradycardia - atropine.
Find out what apoplexy is: causes, symptoms, help, treatment.
Read why oxygen starvation of the brain leads to loss of consciousness. Diagnosis, treatment and consequences of hypoxia.
Everything about epileptic damage to the nervous system: causes, classification of epilepsy, symptoms.
Consequences of the disease
Bettolepsy is a fairly rare phenomenon. This diagnosis is made in approximately 2% of patients who present with similar complaints. Usually attacks do not lead to serious consequences. But this is not a reason to ignore the symptoms and not see a doctor. Since sometimes disturbances in the brain can occur, and with mild seizures, the patient can simply suffer from a fall.
The prognosis is generally favorable. Most often, it is enough to cure the provoking disease, and if this is not possible, then avoid severe coughing attacks. To do this, use antitussive drugs or, for example, breathing exercises.
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Source: https://golmozg.ru/zabolevanie/bettolepsiya.html
What causes symptoms such as: loss of consciousness, cough
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- muscle pain
Whooping cough
Whooping cough is an acute respiratory infection that is bacterial in nature.
The disease is characterized by an infection in the upper respiratory tract, which is manifested by attacks of spasmodic coughing and difficulty breathing. Read more- fever
- stomach ache
- dyspnea
- loss of consciousness
- vomit
- watery stool
- pale skin
- suffocation
- cough
- dehydration
- hot skin
- high blood pressure
- labored breathing
- spasm
- weakness
- muscle weakness
- elevated temperature
- runny nose
- dry cough
- cough with sputum
- cyanosis
Silicosis
Silicosis is the most common and severe type of pneumoconiosis, an occupational lung disease caused by inhalation of dust containing free silicon dioxide.
It is characterized by diffuse growth of connective tissue in the lungs and the formation of characteristic nodules. This foreign tissue reduces the lungs' ability to process oxygen. Silicosis causes a risk of tuberculosis, bronchitis and emphysema. Read more- poor appetite
- fever
- shortness of breath on exertion
- dyspnea
- night sweats
- swollen lymph nodes
- tachycardia
- dizziness
- weight loss
- chest pain
- suffocation
- cough
- headache
- hemoptysis
- heart rhythm disturbances
- irritability
- fatigue
- weakness
- cardiopalmus
- rapid breathing
- chronic fatigue
- respiratory failure
- shallow breathing
- coughing up blood
- asthma attacks
Bronchiolitis
Bronchiolitis is an acute inflammatory process in the bronchioles (the terminal branches of the bronchial tree), leading to difficulty breathing, and in severe cases, suffocation.
The disease is considered a childhood disease, since it most often occurs in children from birth to 3 years. Read more- poor appetite
- dyspnea
- stuffy nose
- tachycardia
- vomit
- fatigue
- weight loss
- cough
- retraction of intercostal spaces
- irritability
- labored breathing
- fatigue
- flaring nostrils
- nausea
- rapid breathing
- elevated temperature
- runny nose
- dry cough
- cough with sputum
- cyanosis
- wheezing in the lungs
Glomerulonephritis
Glomerulonephritis is a serious condition that can interfere with the normal functioning of the kidneys.
Read more- polyuria
- poor appetite
- fever
- seizures
- shortness of breath on exertion
- hiccups
- itchy skin
- insomnia
- drowsiness
- swelling due to excess fluid
- swollen ankle
- tactile hypoesthesia
- swelling
- abdominal pain
- muscle twitching
- loss of consciousness
- unusual color of urine
- dark urine
- vomit
- vomiting blood
- dark vomit
- fatigue
- weight loss
- anemia
- pale skin
- frequent bleeding
- chills
- coma
- contusion
- decreased attention
- rave
- hot skin
- headache
- hematuria
- gastrointestinal bleeding
- high blood pressure
- increased skin pigmentation
- tendency to bruise
- fatigue
- lethargy
- lower back pain
- confusion
- spasm
- nocturia
- nosebleed
- bad feeling
- lack of appetite
- oliguria
- painful urination
- weakness
- nausea
- elevated temperature
- sweating
- urine with blood
- red urine color
- infrequent urination
Severe acute respiratory syndrome
Severe acute respiratory syndrome (SARS) is a serious form of viral pneumonia. Severe acute respiratory syndrome (SARS), better known as SARS, purple death, also in the media - atypical pneumonia - the first case of the disease was registered in November 2002 in the Chinese province of Guangdong. The causative agent is the SARS coronavirus, which belongs to the subfamily of coronaviruses. During the process of reproduction, the virus destroys the cells of the pulmonary alveoli.
- poor appetite
- fever
- stomach ache
- rash
- dyspnea
- night sweats
- a sore throat
- tremor
- vomit
- watery stool
- woozy
- chills
- cough
- hot skin
- headache
- confusion
- myalgia
- bad feeling
- pain
- painful breathing
- weakness
- diarrhea
- elevated temperature
- body aches
- dry cough
- breathing disorder
Goodpasture's syndrome
Goodpasture syndrome is an autoimmune disease that affects small blood vessels in the kidneys and lungs because the body produces autoantibodies to them.
Accompanied by pulmonary hemorrhage, people with weak immune systems are most susceptible, and is quite rare. Read more- poor appetite
- severe blood loss
- dyspnea
- dark urine
- vomit
- fatigue
- weight loss
- pale skin
- bloody sputum
- chest pain
- cough
- hematuria
- hemoptysis
- high blood pressure
- joint pain
- fatigue
- lethargy
- lack of appetite
- oliguria
- pain
- weakness
- nausea
- elevated temperature
- dry cough
- muscle pain
- swelling of the legs
- cyanosis
- wheezing in the lungs
- urine with blood
- swelling of the hands
- foamy urine
- burning sensation when urinating
- back pain below the ribs
- coughing up blood
Beck's sarcoid
Beck's sarcoid is a common skin disorder.
This problem is a type of cutaneous sarcoidosis. This disease is characterized by the appearance of granulomatous growths of a lumpy or red color, which are localized on the face, extensor limbs, upper back, oral mucosa, and in the ears. The peculiarity of the disease is that in its presence, tuberculosis reactions are negative, and the Kveim reaction (intradermal injection of sarcoidosis antigen) is positive. Read more- poor appetite
- fever
- rash
- dyspnea
- blurred vision
- weight loss
- chest pain
- hemoptysis
- joint pain
- lethargy
- apathy
- muscle weakness
- elevated temperature
- dry cough
- muscle pain
- chronic fatigue
- rash on face
Ascariasis
Ascariasis is a helminthic infestation caused by the roundworm Ascaris lumbricoides.
This helminth has a spindle-shaped shape, white-pink color and large sizes (males - about 25 cm, females - about 40 cm). Read more- poor appetite
- bad memory
- fever
- itchy skin
- rash
- dyspnea
- wheeze
- bloating
- swollen lymph nodes
- abdominal pain
- vomit
- watery stool
- fatigue
- weight loss
- headache
- irritability
- fatigue
- liver enlargement
- weakness
- abdominal pain
- nausea
- chest discomfort
- increased salivation
- dry cough
- cough with sputum
- enlarged spleen
Hypercalcemia
Hypercalcemia is an increase in the concentration of calcium in the blood plasma.
With a high concentration of calcium, a calcium deposit forms in the blood vessels, liver and kidney tissue, which makes them brittle. Read more- polydipsia
- poor appetite
- bad memory
- problem behavior
- seizures
- stomach ache
- pain in the side
- drowsiness
- spinal deformity
- abdominal pain
- muscle twitching
- constipation
- loss of consciousness
- vomit
- weak muscles
- fatigue
- coma
- dehydration
- rave
- dementia
- depression
- dry skin
- hallucinations
- headache
- hematuria
- high blood pressure
- irritability
- fatigue
- apathy
- memory loss
- confusion
- muscle atony
- nocturia
- lack of appetite
- pain
- nausea
- thirst
- frequent urination
- muscle pain
Echinococcosis
Echinococcosis is a popular disease caused by parasites.
The greatest prevalence of the problem is noted in countries where agriculture is developed. The infection is usually found in the feces of animals (mainly dogs and rodents). In the intestines, helminth eggs hatch into larvae that enter the blood vessels and transform there into echinococcal cysts (Finns). Echinococcus can affect any organs, but in most cases the following types of disease occur: Read more- poor appetite
- abdominal pain
- dizziness
- vomit
- weight loss
- chest pain
- chills
- headache
- fatigue
- weakness
- nausea
- diarrhea
- elevated temperature
- decreased performance
- recurrent headaches
- skin rashes
- dry cough
- pain in the right hypochondrium
- red spots
Chronic kidney disease
Chronic kidney disease (CKD) is progressive and irreversible damage to the kidneys. The most common causes of chronic kidney disease are high blood pressure and diabetes.
- polydipsia
- poor appetite
- rectal bleeding
- seizures
- impotence
- stomach ache
- hiccups
- itchy skin
- insomnia
- smell from the mouth
- dyspnea
- drowsiness
- swollen ankle
- tactile hypoesthesia
- swelling
- muscle twitching
- restlessness
- vomit
- fatigue
- weight loss
- pale skin
- frequent bleeding
- coma
- dehydration
- rave
- headache
- high blood pressure
- increased skin pigmentation
- tendency to bruise
- lethargy
- apathy
- confusion
- spasm
- bad nails
- nocturia
- bad feeling
- lack of appetite
- weakness
- nausea
- thirst
- diarrhea
- frequent urination
- yellow skin
- itching
- dry mouth
- swelling of the legs
- swelling of the hands
Never rely solely on search results to make a diagnosis. Be sure to consult a board-certified physician for diagnosis and treatment.
Cough until you lose consciousness what to do
Bettolepsy is a neurological disorder. It is characterized by impaired consciousness during a severe coughing attack. Bettolepsy refers to cough syncope (syncope is a short-term loss of consciousness). Most often, the disorder occurs in men over 40 years of age.
Causes
Cough syncope occurs due to disorders of the internal organs:
- Pulmonary diseases: bronchial asthma, cor pulmonale, tuberculosis and emphysema. With pulmonary diseases, blood stagnates in the pulmonary circulation, which causes cardiopulmonary failure.
- Diseases and pathological conditions of the upper respiratory tract: whooping cough, foreign bodies entering the bronchi. During attacks, oxygen does not reach the lungs. Hypoxia of the brain occurs, the person loses consciousness.
- Brain pathologies: dyscirculatory encephalopathy, arteriovenous malformations, previous traumatic brain injuries, increased intracranial pressure.
- Peripheral nervous system disorders. When the laryngeal nerve is inflamed, the vagus nerve is activated due to the generation of a large number of electrical impulses. The number of heart contractions decreases and cardiac output decreases. The brain receives less blood and the person loses consciousness.
These risk factors increase the likelihood of developing betolepsy:
- smoking;
- sedentary lifestyle;
- diet without vegetables and fruits;
- alcoholism, drug addiction.
The exact pathophysiological mechanisms are unknown. However, the hemodynamic theory has the most supporters. To understand the mechanism of development of cough syncope, you need to understand the mechanism of cough. It consists of three acts:
The first act is inspiratory. Characterized by closure of the larynx. The second act is compression: the muscles of the chest and abdomen contract, the diaphragm is fixed in a static position. The third act is expiratory: high pressure is created, which pushes air out at high speed with the larynx open.
In the second and third phases, intrathoracic pressure increases. With a normal cough, the pressure increases from 40 to 100 mm Hg. During syncopal cough, the pressure tends to 200-300 mm Hg. Due to such indicators, intra-abdominal pressure increases.
This results in decreased blood flow to the heart. As a result, the amount of blood ejected by the heart decreases and systolic blood pressure decreases. Blood flow to the brain decreases. In neurons – hypoxia and ischemia.
Due to lack of oxygen to the cortex, a person loses consciousness.
There are continuous and intermittent coughs. In the first option, a coughing attack occurs that lasts several seconds. Between acts of coughing, a person cannot inhale air. Because of this, the number of impulses to baroreceptors decreases, and the resistance of peripheral vessels reflexively increases.
With an intermittent cough, a person inhales air before each coughing act. This irritates the baroreceptors and decreases peripheral resistance.
These mechanisms lead to difficulty in venous outflow from the brain. Intracranial pressure increases. The minute volume of blood circulation in the cerebral arteries decreases. The bleeding may stop completely. Due to hypoxia, syncope occurs.
Symptoms
The clinical picture of bettolepsy is considered from 4 positions:
- The pathology is often observed in mature and elderly men who smoke and are obese. Often combined with bronchopulmonary diseases.
- Loss of consciousness is most often caused by chronic paroxysmal cough. Coughing is accompanied by strong contraction of the muscles of the chest and abdomen.
- Syncope can develop in any body position: lying, standing or sitting. Usually a person loses consciousness after 5 seconds of continuous coughing. Loss of consciousness is preceded by dizziness and decreased visual accuracy.
- Syncope due to continuous coughing lasts up to 10 seconds, in rare cases, loss of consciousness lasts 3 minutes. Syncope may be accompanied by cramps and heavy sweating. Some patients experience involuntary urination or defecation.
Diagnosis and treatment
To diagnose bettolepsy, it is usually enough for a doctor to study the history of life and illness, since the symptoms of syncopal cough are specific - they are difficult to confuse with signs of another disease. To clarify the diagnosis, doctors prescribe vagal tests, electroencephalography, and assessment of the functioning of the bronchi and lungs.
A person with an attack of bettolepsy needs first aid. A patient in syncope needs to ensure blood flow to the brain. To do this, lay the person on his back, raise his legs and lower his head. It is also recommended to open the window and relieve the upper body: unbutton your shirt, take off your tie.
To eliminate bettolepsy, you need to cure the underlying disease that causes attacks of suffocating cough. The patient also needs to quit smoking, lose excess weight and engage in physical activity.
Bettolepsy cannot be treated with folk remedies, since the underlying disease can progress and more often cause fainting.
Bettolepsy (Laryngeal vertigo, Cough-brain syndrome, Cough syncope, Cough syncope, Respiratory seizure)
Bettolepsy
- These are transient disturbances of consciousness that occur at the peak of a coughing attack. The syndrome is manifested by cough syncope: short-term twilight consciousness, fainting or deep loss of consciousness, sometimes accompanied by convulsions, involuntary urination and defecation.
Methods for diagnosing bettolepsy include questioning, examination of the patient, functional tests, instrumental studies (electrocardiography, electroencephalography, bronchoscopy).
Treatment involves symptomatic therapy that alleviates the patient’s condition and is aimed at eliminating the manifestations of the underlying disease.
Causes of bettolepsy
The condition occurs against the background of acute or chronic hypoxia of brain tissue. Its immediate cause is a sharp worsening of an already existing lack of oxygen caused by coughing paroxysm. Pathology can manifest itself in the following diseases:
- Chronic pulmonary pathologies
(cor pulmonale, asthma, tuberculosis, emphysema). With these diseases, stagnation occurs in the pulmonary circulation, and pulmonary heart failure subsequently develops. In a decompensated course, the development of encephalopathy with a tendency to convulsive fainting is possible. - Airway obstruction
(aspiration of foreign bodies, whooping cough, acute laryngitis). Accompanied by acute cerebral hypoxia and prolonged bouts of severe coughing, which cause episodes of cough syncope. - Cerebrovascular disorders
. Changes in cerebral vessels (vascular malformations, compression of intracranial and extracranial veins, consequences of head injury) cause venous hyperemia of the brain, which can be accompanied by fainting attacks. Disturbances in the blood supply to the brain due to pathology of extra- and intracranial arteries (cerebral atherosclerosis, vertebral artery syndrome) threaten the development of a number of vestibular disorders, including loss of consciousness. - Lesions of peripheral nerves
. With neuralgia of the superior laryngeal nerve, pathological impulses lead to activation of the vagus nerve center and bradycardia. The volume of cardiac output sharply decreases, cerebral ischemia and fainting occur.
Risk factors for the development of attacks of impaired consciousness are smoking, drug addiction, and excess weight. When intoxicated with alcohol and drugs, changes occur in the brain, its membranes and cerebrospinal fluid, leading to disruption of the respiratory and cardiovascular systems.
Symptoms of bettolepsy
Clinical manifestations may differ not only in different patients, but also each attack in an individual patient can take on different course options. Paroxysmal conditions - cough syncope - occur at the peak of the cough reflex.
Similar symptom complexes are also observed when laughing, sneezing, straining, lifting heavy objects, etc.
They may be preceded by prodromal phenomena (presyncopal states) in the form of dizziness, tinnitus, blurred vision, facial flushing, which is subsequently replaced by cyanosis, and swelling of the neck veins when coughing. In some cases, some warning signs may be missing.
Source: https://cardiotherapy.ru/polezno-znat/kashel-do-poteri-soznaniya-chto-delat
Bettolepsy symptoms and treatment with folk remedies
08.04.2019
Bettolepsy is a neurological disorder. It is characterized by impaired consciousness during a severe coughing attack. Bettolepsy refers to cough syncope (syncope is a short-term loss of consciousness). Most often, the disorder occurs in men over 40 years of age.
Causes
Cough syncope occurs due to disorders of the internal organs:
- Pulmonary diseases: bronchial asthma, cor pulmonale, tuberculosis and emphysema. With pulmonary diseases, blood stagnates in the pulmonary circulation, which causes cardiopulmonary failure.
- Diseases and pathological conditions of the upper respiratory tract: whooping cough, foreign bodies entering the bronchi. During attacks, oxygen does not reach the lungs. Hypoxia of the brain occurs, the person loses consciousness.
- Brain pathologies: dyscirculatory encephalopathy, arteriovenous malformations, previous traumatic brain injuries, increased intracranial pressure.
- Peripheral nervous system disorders. When the laryngeal nerve is inflamed, the vagus nerve is activated due to the generation of a large number of electrical impulses. The number of heart contractions decreases and cardiac output decreases. The brain receives less blood and the person loses consciousness.
These risk factors increase the likelihood of developing betolepsy:
- smoking;
- sedentary lifestyle;
- diet without vegetables and fruits;
- alcoholism, drug addiction.
The exact pathophysiological mechanisms are unknown. However, the hemodynamic theory has the most supporters. To understand the mechanism of development of cough syncope, you need to understand the mechanism of cough. It consists of three acts:
The first act is inspiratory. Characterized by closure of the larynx. The second act is compression: the muscles of the chest and abdomen contract, the diaphragm is fixed in a static position. The third act is expiratory: high pressure is created, which pushes air out at high speed with the larynx open.
In the second and third phases, intrathoracic pressure increases. With a normal cough, the pressure increases from 40 to 100 mm Hg. During syncopal cough, the pressure tends to 200-300 mm Hg. Due to such indicators, intra-abdominal pressure increases.
This results in decreased blood flow to the heart. As a result, the amount of blood ejected by the heart decreases and systolic blood pressure decreases. Blood flow to the brain decreases. In neurons – hypoxia and ischemia.
Due to lack of oxygen to the cortex, a person loses consciousness.
There are continuous and intermittent coughs. In the first option, a coughing attack occurs that lasts several seconds. Between acts of coughing, a person cannot inhale air. Because of this, the number of impulses to baroreceptors decreases, and the resistance of peripheral vessels reflexively increases.
With an intermittent cough, a person inhales air before each coughing act. This irritates the baroreceptors and decreases peripheral resistance.
These mechanisms lead to difficulty in venous outflow from the brain. Intracranial pressure increases. The minute volume of blood circulation in the cerebral arteries decreases. The bleeding may stop completely. Due to hypoxia, syncope occurs.
Symptoms
The clinical picture of bettolepsy is considered from 4 positions:
- The pathology is often observed in mature and elderly men who smoke and are obese. Often combined with bronchopulmonary diseases.
- Loss of consciousness is most often caused by chronic paroxysmal cough. Coughing is accompanied by strong contraction of the muscles of the chest and abdomen.
- Syncope can develop in any body position: lying, standing or sitting. Usually a person loses consciousness after 5 seconds of continuous coughing. Loss of consciousness is preceded by dizziness and decreased visual accuracy.
- Syncope due to continuous coughing lasts up to 10 seconds, in rare cases, loss of consciousness lasts 3 minutes. Syncope may be accompanied by cramps and heavy sweating. Some patients experience involuntary urination or defecation.
Diagnosis and treatment
To diagnose bettolepsy, it is usually enough for a doctor to study the history of life and illness, since the symptoms of syncopal cough are specific - they are difficult to confuse with signs of another disease. To clarify the diagnosis, doctors prescribe vagal tests, electroencephalography, and assessment of the functioning of the bronchi and lungs.
A person with an attack of bettolepsy needs first aid. A patient in syncope needs to ensure blood flow to the brain. To do this, lay the person on his back, raise his legs and lower his head. It is also recommended to open the window and relieve the upper body: unbutton your shirt, take off your tie.
To eliminate bettolepsy, you need to cure the underlying disease that causes attacks of suffocating cough. The patient also needs to quit smoking, lose excess weight and engage in physical activity.
Bettolepsy cannot be treated with folk remedies, since the underlying disease can progress and more often cause fainting.
Didn't find a suitable answer? Find a doctor and ask him a question!
Source: https://sortmozg.com/psihicheskie-rasstrojstva/bettolepsiya
Loss of consciousness when coughing in men
Cases of bettolepsy are quite rare and are observed in no more than 2% of patients with various types of paroxysmal conditions. More often, this syndrome is observed in older men suffering from chronic diseases of the respiratory system.
At a younger age, cough fainting is observed very rarely and is associated with insufficiency of the mechanisms responsible for maintaining postural tone, or increased sensitivity of the carotid sinus. In children, bettolepsy can develop against the background of whooping cough.
In this article we will introduce you to the causes, symptoms and methods of diagnosing and treating this pathology. This information will be useful for you, and you will be able to consult a doctor in time if you suspect the onset of bettolepsy in yourself or your loved ones.
Diagnostics
If attacks of bettolepsy occur, the patient must contact his local doctor, who will refer him for a consultation with a neurologist.
To make a diagnosis, a detailed analysis of the patient’s medical history and life is carried out, the nature of the attacks is carefully studied and an examination plan is drawn up, which allows one to identify the cause of coughing fainting and carry out a differential diagnosis of bettolepsy with other diseases (for example, epilepsy).
To identify cough-brain syndrome, the following types of examinations may be prescribed:
- Valsalva maneuver;
- ECG;
- Holter monitoring;
- EEG;
- blood pressure measurement;
- Echo-CG;
- electrophysiological methods of intracardiac stimulation, etc.
In some cases, patients are advised to undergo tracheobronchoscopy.
The need for hospitalization of a patient in a hospital for examination and treatment is determined individually and depends on the possibility of identifying the causes of the development of bettolepsy in an outpatient treatment facility and the severity of the attacks. Sometimes, if the cause of consciousness disorders is unclear, the patient is advised to undergo examination at a specialized epileptological center.
Source: https://golovnoj-mozg.ru/voprosy/poterya-soznaniya-pri-kashle-u-muzhchin